pro-inflammatory cytokines Archives

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Canna~Fangled Abstracts

Cannabidiol prevents chemotherapy-induced neuropathic pain by modulating spinal TLR4 via endocannabinoid system activation

J Pharm Pharmacol. 2023 Mar 22;rgad023. doi: 10.1093/jpp/rgad023. Online ahead of print. Rafaela Dos Santos 1, Flávio…

Freedom WaresMarch 22, 2023

Canna~Fangled Abstracts

The independent and combined effects of cannabis use and systemic inflammation during the early stages of psychosis: exploring the two-hit hypothesis

Immune dysregulation might be part of the pathophysiology of psychosis, not explained by cannabis use…

Freedom WaresMarch 20, 2021

Canna~Fangled Abstracts

Cannabinoids as Key Regulators of Inflammasome Signaling: A Current Perspective

The importance of cannabinoids as possible drugs to target inflammasome activation to support the treatment…

Freedom WaresJanuary 28, 2021

Canna~Fangled Abstracts

Natural cannabinoids suppress the cytokine storm in sepsis-like in vitro model

Eur Cytokine Netw. 2020 Jun 1;31(2):50-58. doi: 10.1684/ecn.2020.0445. Yishay Szekely 1, Merav Ingbir 1, Ohad S Bentur 1, Ohad Hochner 1, Reuven Porat 1…

Freedom WaresJune 16, 2020

Canna~Fangled Abstracts

Cannabidiol decreases bone resorption by inhibiting RANK/RANKL expression and pro-inflammatory cytokines during experimental periodontitis in rats.

2009 Feb;9(2):216-22. doi: 10.1016/j.intimp.2008.11.010. Epub 2008 Dec 12. Napimoga MH1, Benatti BB, Lima FO, Alves PM, Campos AC, Pena-Dos-Santos DR, Severino…

Freedom WaresFebruary 9, 2009

pro-inflammatory cytokines

Cannabidiol prevents chemotherapy-induced neuropathic pain by modulating spinal TLR4 via endocannabinoid system activation

The independent and combined effects of cannabis use and systemic inflammation during the early stages of psychosis: exploring the two-hit hypothesis

Cannabinoids as Key Regulators of Inflammasome Signaling: A Current Perspective

Natural cannabinoids suppress the cytokine storm in sepsis-like in vitro model

Cannabidiol decreases bone resorption by inhibiting RANK/RANKL expression and pro-inflammatory cytokines during experimental periodontitis in rats.

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